As we age, calcium accumulates in the soft tissue. As
a matter of fact, the age of an organism can be determined by the
amount of this pathological calcification of the soft tissues. In
muscles cells, calcium is the trigger for contraction. As calcium
enters the cell, the muscle cell contracts, and then as it is pumped
out again, the muscle relaxes. If the pathological calcium in a cell
reaches a certain point, and the cell is no longer able to remove
it, then the muscle cell will stay in a contracted position
indefinitely. This is the definition of a trigger point, a
pathologically contracted group of muscle cells that cannot release.
As we age, our muscles become tighter and tighter with more trigger
points becoming evident. These trigger points can also pull the
vertebra out of alignment causing subluxations and compression of
the spinal nerves. Fibromyalgia is the pathological accumulation of
trigger points in a person whose age does not justify the
calcification. Rigor mortis is the ultimate expression of this
muscle contraction. As all ATP production stops in the cells at
death, calcium floods into all the muscle cells and causes global
Various mechanisms of how this happens have been proposed. Some
claim that the kidney's inability to effectively remove phosphorous
from the bloodstream results in a accumulation of acidic phosphorous
in the cells. The body then imports calcium into the cell to
maintain a proper ph. The beneficial results with Guanifenesin
support this claim.
Some suggest that magnesium deficiency lowers ATP production so that
the cells cannot rid themselves of the calcium that naturally enters
the cell due to concentration gradients (1:10,000)
Others suggest infections as a cause. Certain infections cause
hypercoagulation which decreases local circulation, thereby lowering
local oxygen levels. Lower oxygen levels decreases ATP production
and ATP is required to operate the pumps which keep calcium from
accumulating in the cell.
Regardless of the initial cause of Fibromyalgia, the calcium must be
removed to effect a recovery. For this reason, EDTA chelation is an
option since it is able to chelate pathological calcium out of the
The 3 potential causes of Fibromyalgia may also be addressed with
1) Phosphorous accumulation due to kidney insufficiency:
EDTA chelation has been shown to have a normalizing effect on kidney
function bringing low creatine levels up and high creatine levels
down. This information can be found in the following study: "The
effect of EDTA chelation plus supportive multivitamin/trace mineral
supplementation upon renal function. A study in serum creatine. E.
W. McDonagh, D.O."
Also, the increase of metabolic potassium in magnesium di-potassium
EDTA helps displace cellular phosphorous.
2) Low ATP production due to low magnesium levels:
Magnesium is a difficult mineral to absorb and not commonly found in
adequate amounts in the standard American diet. If magnesium based
EDTA is used, then magnesium levels can be restored.
3) Hypercoagulation due to infection:
Chelation is known to reduce hypercoagulation due to its stimulating
effects on prostacyclin and it's inhibitory effects on thromboxane
(the hormones which control the blood clotting cascade).
Thus magnesium based chelation not only addresses the manifestations
of fibromyalgia (the calcifications) but also the potential causes